| [1] Hanahan D,Weinberg RA. Hallmarks of cancer:the next generation[J].Cell,2011,144(5):646-674.[2] Maletzki C,Emmrich J.Inflammation and immunity in the tumor environment[J].Dig Dis,2010,28(4/5):574-578.[3] Di Maggio FM,Minafra L,Forte GI,et al.Portrait of inflammatory response to ionizing radiation treatment[J].J Inflamm (Lond),2015,12(1):12-14.[4] Russell JS,Brown JM.The irradiated tumor microenvironment:role of tumor-associated macrophages in vascular recovery[J].Front Physiol,2013,4(7):157-161.[5] Chanmee T,Ontong P,Konno K,et al.Tumor-associated macrophages as major players in the tumor microenvironment[J].Cancers (Basel),2014,6(3):1670-1690.[6] Pons I,Gras G,Courberand S,et al.Consequences of gamma-irradiation on inflammatory cytokine regulation in human monocytes/macrophages[J].Int J Radiat Biol,1997,71(2):157-166.[7] Conrad S,Ritter S,Fournier C,et al.Differential effects of irradiation with carbon ions and X-rays on macrophage function[J].J Radiat Res,2009,50(3):223-231.[8] Dong C,He M,Tu W,et al.The differential role of human macrophage in triggering secondary bystander effects after either gamma-ray or carbon beam irradiation[J].Cancer Lett,2015,363(1):92-100.[9] Lee SC,Srivastava RM,Lopez-Albaitero A,et al.Natural killer (NK):dendritic cell (DC) cross talk induced by therapeutic monoclonal antibody triggers tumor antigen-specific T cell immunity[J].Immunol Res,2011,50(2/3):248-254.[10] Buchser WJ,Laskow TC,Pavlik PJ,et al.Cell-mediated autophagy promotes cancer cell survival[J].Cancer Res,2012,72(12):2970-2979.[11] Uchida A,Mizutani Y,Nagamuta M,et al.Effects of X-ray irradiation on natural killer (NK) cell system.Ⅱ.Increased sensitivity to natural killer cytotoxic factor (NKCF)[J].Immunopharmacol Immunotoxicol,1989,11(2/3):521-534.[12] Miller GM,Andres ML,Gridley DS.NK cell depletion results in accelerated tumor growth and attenuates the antitumor effect of total body irradiation[J].Int J Oncol,2003,23(6):1585-1592.[13] Barreira DSR,Munz C.Natural killer cell activation by dendritic cells:balancing inhibitory and activating signals[J].Cell Mol Life Sci,2011,68(21):3505-3518.[14] Dong H,Bullock TN.Metabolic influences that regulate dendritic cell function in tumors[J].Front Immunol,2014,5:24-28.[15] Liu C,Lin J,Zhao L,et al.Gamma-ray irradiation impairs dendritic cell migration to CCL19 by down-regulation of CCR7 and induction of cell apoptosis[J].Int J Biol Sci,2011,7(2):168-179.[16] Shigematsu A,Adachi Y,Koike-Kiriyama N,et al.Effects of low-dose irradiation on enhancement of immunity by dendritic cells[J].J Radiat Res,2007,48(1):51-55.[17] Gao F,Liu C,Guo J,et al.Radiation-driven lipid accumulation and dendritic cell dysfunction in cancer[J].Sci Rep,2015,5(4):9613-9618.[18] Qi W,Huang X,Wang J.Correlation between Th17 cells and tumor microenvironment[J].Cell Immunol,2013,285(1/2):18-22.[19] Ye J,Livergood RS,Peng G.The role and regulation of human Th17 cells in tumor immunity[J].Am J Pathol,2013,182(1):10-20.[20] Su Z,Sun Y,Zhu H,et al.Th17 cell expansion in gastric cancer may contribute to cancer development and metastasis[J].Immunol Res,2014,58(1):118-124.[21] Lee EJ,Park HJ,Lee IJ,et al.Inhibition of IL-17A suppresses enhanced-tumor growth in low dose pre-irradiated tumor beds[J].PLoS One,2014,9(9):e106423.[22] Wang Q,Franks HA,Lax SJ,et al.The ataxia telangiectasia mutated kinase pathway regulates IL-23 expression by human dendritic cells[J].J Immunol,2013,190(7):3246-3255.[23] Katz LH,Li Y,Chen JS,et al.Targeting TGF-beta signaling in cancer[J].Expert Opin Ther Targets,2013,17(7):743-760.[24] Moore BB,Murray L,Das A,et al.The role of CCL12 in the recruitment of fibrocytes and lung fibrosis[J].Am J Respir Cell Mol Biol,2006,35(2):175-181.[25] An YS,Kim MR,Lee SS,et al.TGF-beta signaling plays an important role in resisting gamma-irradiation[J].Exp Cell Res,2013,319(4):466-473.[26] Hong GU,Kim NG,Ro JY.Expression of airway remodeling proteins in mast cell activated by TGF-beta released in OVA-induced allergic responses and their inhibition by low-dose irradiation or 8-oxo-dG[J].Radiat Res,2014,181(4):425-438.[27] Li N,Grivennikov SI,Karin M.The unholy trinity:inflammation,cytokines,and STAT3 shape the cancer microenvironment[J].Cancer Cell,2011,19(4):429-431.[28] Li F,Gao L,Jiang Q,et al.Radiation enhances the invasion abilities of pulmonary adenocarcinoma cells via STAT3[J].Mol Med Rep,2013,7(6):1883-1888.[29] Zhang MX,Zhao X,Wang ZG,et al.Constitutive activation of signal transducer and activator of transcription 3 regulates expression of vascular endothelial growth factor in human meningioma differentiation[J].J Cancer Res Clin Oncol,2010,136(7):981-988.[30] Gao L,Li FS,Chen XH,et al.Radiation induces phosphorylation of STAT3 in a dose- and time-dependent manner[J].Asian Pac J Cancer Prev,2014,15(15):6161-6164.[31] Bao B,Thakur A,Li Y,et al.The immunological contribution of NF-kappaB within the tumor microenvironment:a potential protective role of zinc as an anti-tumor agent[J].Biochim Biophys Acta,2012,1825(2):160-172.[32] Guo Z,Xing Z,Cheng X,et al.Somatostatin derivate (smsDX) attenuates the TAM-stimulated proliferation,migration and invasion of prostate cancer via NF-κB regulation[J].PLoS one,2015,10(5):292-307.[33] Bai M,Ma X,Li X,et al.The accomplices of NF-kappaB lead to radioresistance[J].Curr Protein Pept Sci,2015,16(4):279-294.[34] Veuger SJ,Hunter JE,Durkacz BW.Ionizing radiation-induced NF-kappaB activation requires PARP-1 function to confer radioresistance[J].Oncogene,2009,28(6):832-842. |
