[1] Hannafon F,Cadogan MP.Recognition and treatment of postmenopausal osteoporosis[J].J Gerontol Nurs,2014,40(3):10-14. [2] Geusens P.New insights into treatment of osteoporosis in postmenopausal women[J].RMD Open,2015,1(Suppl 1):e000051. [3] Cohen A,Kamanda-Kosseh M,Recker RR,et al.Bone density after teriparatide discontinuation in premenopausal idiopathic osteoporosis[J].J Clin Endocrinol Metab,2015,100(11):4208-4214. [4] Shieh A,Han W,Ishii S,et al.Quantifying the balance between total bone formation and total bone resorption:An index of net bone formation[J].J Clin Endocrinol Metab,2016,101(7):2802-2809. [5] Hu S,Liu CC,Chen G,et al.In vivo effects of two novel ALN-EP4a conjugate drugs on bone in the ovariectomized rat model for reversing postmenopausal bone loss[J].Osteoporos Int,2016,27(2):797-808. [6] McNabb BL,Vittinghoff E,Schwartz AV,et al.BMD changes and predictors of increased bone loss in postmenopausal women after a 5-year course of alendronate[J].J Bone Miner Res,2013,28(6):1319-1327. [7] Jafari A,Qanie D,Andersen TL,et al.Legumain regulates differentiation fate of human bone marrow stromal cells and is altered in postmenopausal ssteoporosis[J].Stem Cell Rep,2017,8(2):373-386. [8] Mujcic H,Nagelkerke A,Rouschop KM,et al.Hypoxic activation of the PERK/eIF2α arm of the unfolded protein response promotes metastasis through induction of LAMP3[J].Clin Cancer Res,2013,19(22):6126-6137. [9] Wei J,Sheng X,Feng D,et al.PERK is essential for neonatal skeletal development to regulate osteoblast proliferation and differentiation[J].J Cell Physiol,2008,217(3):693-707. [10] Yu Y,Pierciey FJ Jr,Maguire TG,et al.PKR-like endoplasmic reticulum kinase is necessary for lipogenic activation during HCMV infection[J].PLoS Pathog,2013,9(4):e1003266. [11] Dizdar M,Irdesel JF,Dizdar OS,et al.Effects of balance-coordination,strengthening and aerobic exercises to prevent falls in postmenopausal patients with osteoporosis:A 6-month randomized parallel prospective study[J].J Aging Phys Act,2018,26(1):41-51. [12] Satpathy S,Patra A,Ahirwar B.Experimental techniques for screening of antiosteoporotic activity in postmenopausal osteoporosis[J].J Complement Integr Med,2015,12(4):251-266. [13] Zhao R,Xie P,Zhang K,et al.Selective effect of hydroxyapatite nanoparticles on osteoporotic and healthy bone formation correlates with intracellular calcium homeostasis regulation[J].Acta Biomater, 2017,59:338-350. [14] Moshiri A,Oryan A,Meimandi-Parizi A.Synthesis,development,characterization and effectiveness of bovine pure platelet gel-collagen-polydioxanone bioactive graft on tendon healing[J].J Cell Mol Med,2015,19(6):1308-1332. [15] Fang Z,Yang Q,Xiong W,et al.Effect of CGRP-adenoviral vector transduction on the osteoblastic differentiation of rat adipose-derived stem cells[J]. PLoS One,2013,8(8):e72738. [16] Choi YH,Han Y,Jin SW,et al.Pseudoshikonin I enhances osteoblast differentiation by stimulating Runx2 and Osterix[J].J Cell Biochem,2018, 119(1):748-757. [17] Xu L,Kong Q.Research progress of key signaling pathways in osteoblast differentiation and bone formation regulation[J].Zhongguo Xiu Fu Chong Jian Wai Ke Za Zhi,2014,28(12):1484-1489. [18] Sun F,Li X,Yang C,et al.A role for PERK in the mechanism underlying fluoride-induced bone turnover[J].Toxicology,2014,325:52-66. [19] Wei J,Sheng X,Feng D,et al.PERK is essential for neonatal skeletal development to regulate osteoblast proliferation and differentiation[J].J Cell Physiol,2008,217(3):693-707. |