[1] 蒋秋梅,晏波,苟静,等.系统性红斑狼疮患者中性粒细胞胞外诱捕网水平分析[J].中华风湿病学杂志,2017,21(8):542-546.
[2] Darrah E, Andrade F. NETs:the missing link between cell death and systemic autoimmune diseases[J].Front Immunol,2012,3:428.
[3] Brinkmann V, Reichard U, Goosmann C, et al. Neutrophil extracellular traps kill bacteria[J]. Science, 2004,303(5663):1532-1535.
[4] Fuchs TA, Abed U,Goosmann C, et al. Novel cell death program leads to neutrophil extracellular traps[J]. J Cell Biol, 2007,176(2):231-241.
[5] Stojkov D, Amini P, Oberson K, et al. ROS and glutathionylation balance cytoskeletal dynamics in neutrophil extracellular trap formation[J]. J Cell Biol,2017,216(12):4073-4090.
[6] Muãoz LE, Kaplan MJ, Radic M, et al. Editorial:NETosis 2:The excitement continues[J]. Front Immunol,2017,8:1318.
[7] Dieker J, Tel J, Pieterse E, et al. Circulating apoptotic microparticles in systemic lupus erythematosus patients drive the activation of dendritic cell subsets and prime neutrophils for NETosis[J]. Arthritis Rheumatol,2016,68(2):462-472.
[8] Truszewska A, Foroncewicz B, Pączek L. The role and diagnostic value of cell-free DNA in systemic lupus erythematosus[J]. Clin Exp Rheumatol,2017,35(2):330-336.
[9] Wang H, Li T, Chen S, et al. Neutrophil extracellular trap mitochondrial DNA and its autoantibody in systemic lupus erythematosus and a proof-of-concept trial of metformin[J]. Arthritis Rheumatol,2015,67(12):3190-3200.
[10] Lande R, Ganguly D, Facchinetti V, et al. Neutrophils activate plasmacytoid dendritic cells by releasing self-DNA-peptide complexes in systemic lupus erythematosus[J]. Sci Transl Med, 2011, 3(73):73ra19.
[11] Urban CF, Ermert D, Schmid M, et al. Neutrophil extracellular traps contain calprotectin, a cytosolic protein complex involved in host defense against Candida albicans[J]. PLoS Pathog,2009,5(10):e100639.
[12] Pieterse E, Hofstra J, Berden J, et al. Acetylated histones contribute to the immunostimulatory potential of neutrophil extracellular traps in systemic lupus erythematosus[J]. Clin Exp Immunol, 2015, 179(1):68-74.
[13] Knight JS, Carmona-Rivera C, Kaplan MJ. Proteins derived from neutrophil extracellular traps may serve as self-antigens and mediate organ damage in autoimmune diseases[J]. Front Immunol, 2012, 3(Suppl):380.
[14] Farrera C, Fadeel B. Macrophage clearance of neutrophil extracellular traps is a silent process[J]. J Immunol,2013,191(5):2647-2656.
[15] Zykova SN, Tveita AA, Rekvig OP. Renal dnase1 enzyme activity and protein expression is selectively shut down in murine and human membranoproliferative lupus nephritis[J]. PLoS One, 2010,5(8):e12096.
[16] Leffler J, Gullstrand B, Jönsen A, et al. Degradation of neutrophil extracellular traps co-varies with disease activity in patients with systemic lupus erythematosus[J]. Arthrit Res Ther,2013,15(4):R84.
[17] Yeh TM, Chang HC, Liang CC, et al. Deoxyribonuclease-inhibitory antibodies in systemic lupus erythematosus[J]. Biomed Sci,2003,10(5):544-551.
[18] Hakkim A, Furnrohr BG, Amann K, et al. Impairment of neutrophil extracellular trap degradation is associated with lupus nephritis[J]. Proc Natl Acad Sci USA,2010,107(21):9813-9818.
[19] Leffler J, Martin M, Gullstrand B, et al. Neutrophil extracellular traps that are not degraded in systemic lupus erythematosus activate complement exacerbating the disease[J]. J Immunol,2012, 188(7):3522-3531.
[20] Mahajan A, Herrmann M, Muñoz LE. Clearance deficiency and cell death pathways:A model for the pathogenesis of SLE[J]. Front Immunol,2016,7:35.
[21] Kahlenberg JM, Carmonarivera C, Smith CK, et al. Neutrophil extracellular trap-associated protein activation of the NLRP3 inflammasome is enhanced in lupus macrophages[J]. J Immunol, 2013, 190(3):1217-1226.
[22] Villanueva E, Yalavarthi S, Berthier CC, et al. Netting neutrophils induce endothelial damage, infiltrate tissues, and expose immunostimulatory molecules in systemic lupus erythematosus[J]. J Immunol,2011,187(1):538-552.
[23] Denny MF, Yalavarthi S, Zhao W, et al. A distinct subset of proinflammatory neutrophils isolated from patients with systemic lupus erythematosus induces vascular damage and synthesizes type Ⅰ interferons[J]. J Immunol,2010,184(6):3284-3297.
[24] Giannelou M, Mavragani CP. Cardiovascular disease in systemic lupus erythematosus:A comprehensive update[J]. J Autoimmun,2017,82:1-12.
[25] Carmona-Rivera C, Zhao W, Yalavarthi S, et al. Neutrophil extracellular traps induce endothelial dysfunction in systemic lupus erythematosus through the activation of matrix metalloproteinase-2[J]. Ann Rheumat Dis,2015,74(7):1417-1424.
[26] Garcia-Romo GS, Caielli S, Vega B, et al. Netting neutrophils are major inducers of type Ⅰ IFN production in pediatric systemic lupus erythematosus[J]. Sci Transl Med, 2011, 3(73):73ra20.
[27] Jimé nez-Alcázar M, Rangaswamy C, Panda R, et al. Host DNases prevent vascular occlusion by neutrophil extracellular traps[J]. Science,2017,358(6367):1202-1206.
[28] Martínez Valle F, Balada E, Ordi-Ros J, et al. DNase 1 and systemic lupus erythematosus[J]. Autoimmun Rev,2008,7(5):359-363.
[29] Lood C, Blanco LP, Purmalek MM, et al. Neutrophil extracellular traps enriched in oxidized mitochondrial DNA are interferogenic and contribute to lupus-like disease[J]. Nat Med, 2016,22(2):146-153.
[30] Van Avondt K,Fritsch-Stork R, Derksen RH, et al. Ligation of signal inhibitory receptor on leukocytes-1 suppresses the release of neutrophil extracellular traps in systemic lupus erythematosus[J]. PLoS One,2013,8(10):e78459.
[31] Knight JS, Subramanian V, O'Dell AA, et al. Peptidylarginine deiminase inhibition disrupts NET formation and protects against kidney, skin and vascular disease in lupus-prone MRL/lpr mice[J]. Ann Rheumat Dis,2015,74(12):2199-2206.
[32] Knight JS, Luo W, O'Dell A A, et al. Peptidylarginine deiminase inhibition reduces vascular damage and modulates innate immune responses in murine models of atherosclerosis[J]. Circulat Res,2014,114(6):947-956.
[33] Remijsen Q, Berghe TV, Wirawan E, et al. Neutrophil extracellular trap cell death requires both autophagy and superoxide generation[J]. Cell Res,2011,21(2):290-304.
[34] Smith CK, Vivekanandan-Giri A, Tang C, et al. Neutrophil extracellular trap-derived enzymes oxidize high-density lipoprotein:An additional proatherogenic mechanism in systemic lupus erythematosus[J]. Arthrit Rheumatol, 2014, 66(9):2532-2544.
[35] Gupta AK, Giaglis S, Hasler P, et al. Efficient neutrophil extracellular trap induction requires mobilization of both intracellular and extracellular calcium pools and is modulated by cyclosporine A[J]. PLoS One,2014,9(5):e97088.
[36] Handono K, Sidarta YO, Pradana BA, et al. Vitamin D prevents endothelial damage induced by increased neutrophil extracellular traps formation in patients with systemic lupus erythematosus[J]. Acta Med Indones, 2014, 46(3):189-198. |
