[1] Broadhead ML, Clark JC, Dass CR, et al.Therapeutic targeting of osteoclast function and pathways [J].Expert Opin Ther Targets, 2011, 15(2):169-181.[2] Jiang L, Tao C, He A, et al.Overexpression of miR-126 sensitizes osteosarcoma cells to apoptosis induced by epigallocatechin-3-gallate [J].World J Surg Oncol, 2014, 12:383.[3] Tian X, Zhang J, Yan L, et al.MiRNA-15a inhibits proliferation, migration and invasion by targeting TNFAIP1 in human osteosarcoma cells[J].Int J Clin Exp Pathol, 2015, 8(6):6442-6449.[4] Liu SH, Zhao JH, Deng KK, et al.Effect of radiation on cytotoxicity, apoptosis and cell cycle arrest of human osteosarcoma MG-63 induced by a ruthenium(Ⅱ) complex[J].Spectrochim Acta A Mol Biomol Spectrosc, 2015, 140:202-209.[5] Duarte IF, Lamego I, Marques J, et al.Nuclear magnetic resonance (NMR) study of the effect of cisplatin on the metabolic profile of MG-63 osteosarcoma cells[J].J Proteome Res, 2010, 9(11):5877-5886.[6] Ye F, Wang H, Zhang L, et al.Baicalein induces human osteosarcoma cell line MG-63 apoptosis via ROS-induced BNIP3 expression[J]. Tumour Biol, 2015[Epub ahead of print].[7] 王秀力, 李春梅, 吕 茜, 等.胰岛素样生长因子1诱导骨髓间充质干细胞分化为心肌样细胞的实验研究[J].中华心血管病杂志, 2013, 41(2): 150-155.[8] Fang D, Yang H, Lin J, et al.17β-estradiol regulates cell proliferation, colony formation, migration, invasion and promotes apoptosis by upregulating miR-9 and thus degrades MALAT-1 in osteosarcoma cell MG-63 in an estrogen receptor-independent manner[J].Biochem Biophys Res Commun, 2015, 457(4):500-506.[9] Huang XC, Maimaiti XY, Huang CW, et al.Synergistic effects of arsenic trioxide combined with ascorbic acid in human osteosarcoma MG-63 cells: a systems biology analysis[J].Eur Rev Med Pharmacol Sci, 2014, 18(24):3877-3888.[10] Ma J, Huang H, Han Z, et al.RLN2 is a positive regulator of AKT-2-induced gene expression required for osteosarcoma cells invasion and chemoresistance[J].Biomed Res Int, 2015, 2015:147468.[11] Dai JQ, Huang YG, He AN.Dihydromethysticin kavalactone induces apoptosis in osteosarcoma cells through modulation of PI3K/Akt pathway, disruption of mitochondrial membrane potential and inducing cell cycle arrest[J].Int J Clin Exp Pathol, 2015,8(5):4356-4366.[12] 刘 勇, 郑志超, 赵 岩, 等.弥漫浸润型胃癌患者术前多西紫杉醇+顺铂+氟尿嘧啶方案化疗的疗效及安全性观察[J].实用医院临床杂志, 2015,12(2): 65-68.[13] 陈泽程, 邓燕明, 冯卫能, 等.5-氟尿嘧啶、顺铂联合多西紫杉醇治疗晚期复发鼻咽癌临床观察[J].现代医药卫生, 2015,31(6): 903-905.[14] Vanoevelen J, Janssens A, Huitema LF, et al.Trpv5/6 is vital for epithelial calcium uptake and bone formation[J].The FASEB J, 2011, 25(9):3197-3207.[15] Fan H, Shen YX, Yuan YF.Expression and prognostic roles of TRPV5 and TRPV6 in non-small cell lung cancer after curative resection[J].Asian Pac J Cancer Prev, 2014,15(6):2559-2563. |