| [1] Alzheimer A.Uber eine eigenartige Erkrankung der Hirnrinde [J].Allg Z Psychiat Psych-Gericht Med,1907,64:146-148.[2] Jack CR Jr,Albert MS,Knopman DS,et al.Introduction to the recommendations from the National Institute on Aging-Alzheimer's Association workgroups on diagnostic guidelines for Alzheimer's disease[J].Alzheimers Dement,2011,7(3):257-262.[3] Ballard C,Gauthier S,Corbett A,et al.Alzheimer's disease [J]. Lancet,2011,377(9770):1019-1031.[4] Bohnen N,Warner MA,Kokmen E,et al. Early and midlife exposure to anesthesia and age of onset of Alzheimer's disease[J]. Int J Neurosci,1994,77(3/4):181-185.[5] Wang Q,Liang G,Yang H,et al. The common inhaled anesthetic isoflurane increases aggregation of huntingtin and alters calcium homeostasis in a cell model of Huntington's disease[J]. Toxicol Appl Pharmacol,2011,250(3):291-298.[6] Bianchi SL,Tran T,Liu C, et al. Brain and behavior changes in 12-month old Tg2576 and nontransgenic mice exposed to anesthetics[J]. Neurobiol Aging,2008,29(7):1002-1010.[7] Perucho J,Casarejos MJ,Gomez A,et al. Trehalose protects from aggravation of amyloid pathology induced by isoflurane anesthesia in APP (swe) mutant mice [J].Curt Alzheimer Res,2012,9(3):334-343.[8] Jing Du,Yu Liang,Feng Xu,et al. Trehalose rescues Alzheimer's disease phenotypes in APP/PS1 transgenic mice [J]. J Pharm Pharmacol,2013,65(12):1753-1756.[9] Rönnbäck A,Pavlov PF,Mansory M,et al. Mitochondrial dysfunction in a transgenic mouse model expressing human amyloid precursor protein (APP) with the Arctic mutation [J]. J Neurochem,2016,136(3):497-502.[10] Borza LR.A review on the cause-effect relationship between oxidative stress and toxic proteins in the pathogenesis of neurodegenerative diseases [J]. Rev Med Chir Soc Med Nat Iasi,2014,118 (1):19-27.[11] Li F,Yang Z,Lu Y,et al. Malondialdehyde suppresses cerebral function by breaking homeostasis between excitation and inhibition in turtle Trachemys scripta [J]. PLoS One,2010,5(12):e15325.[12] Guix FX,Ill-Raga G,Bravo R,et al. Amyloid dependent triosephosphate isomerase nitrotyrosination induces glycation and tau fibrillation [J]. Brain,2009,132(5):1335-1345.[13] Butterfield DA. The 2013 SFRBM discovery award:selected discoveries from the butterfield laboratory of oxidative stress and its sequela in brain in cognitive disorders exemplified by Alzheimer disease and chemotherapy induced cognitive Impairment [J]. Free Radic Biol Med,2014,74(1):157-174.[14] Gu X,Sun J,Li S,et al. Oxidative stress induces DNA demethylation and histone acetylation in SH-SY5Y cells:potential epigenetic mechanisms in gene transcription in Aβ Production [J]. Neurobiol Aging,2013,34(4):1069-1079.[15] Zuo L,Hemmelgarn BT,Chuang CC,et al. The role of oxidative stress induced epigenetic alterations in amyloid-β production in Alzheimer's disease [J]. Oxid Med Cell Longev,2015,2015:1-13.[16] Szarka A. The role of β-amyloid and mitochondrial dysfunction inthe pathogenesis of Alzheimer's disease [J]. Ideggyogy Sz,2015,68(7/8):222-228.[17] 梁春荣,刘雨辉,王叶冉,等.阿尔茨海默病患者外周血炎症因子水平与认知功能的相关性研究[J].解放军医学杂志,2014,39(2):133-137.[18] Emanuele E. Can trehalose prevent neurodegeneration? Insights from experimental studies [J]. Curr Drug Targets,2014,15(5):551-557.[19] Luyckx J,Baudouin C. Trehalose:an intriguing disaccharide with potential for medical application in ophthalmology [J]. Clin Ophthalmol,2011,5:577-581. |
